Treatment Option for Diabetes Type II

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Diabetes and Drug Treatments

TYPE II DIABETES AND DRUG TREATMENTS

Differences

Type I Diabetes

Diabetes mellitus type 1 result from autoimmune destruction of insulin-producing beta cells within the pancreas. Subsequent shortage of insulin translates to increased urine and blood glucose. The developed world is associated with increasingly variant type I diabetes with a large cause of kidney failure and non-traumatic blindness. The link is based on an increased dementia and cognitive dysfunction risk through disease processes of vascular dementia and Alzheimer's disease. The complications are inclusive of sexual dysfunction, acanthosis nigricans, and frequent infections (Jones, et al., 2012).

Type II Diabetes

The classic diabetes symptoms include polyuria (frequent urination), increased thirst, increased hunger (polyphagia), and weight loss. Alternative symptoms commonly presented in the diagnosis include histories of blurred vision, fatigue, recurrent vaginal infections, peripheral neuropathy, and itchiness (Barnett, 2012). However, many people lack symptoms in the initial years and while the diagnosis is presented to routine testing. People suffering from type II diabetes mellitus rarely present hyperosmolar hyperglycemic states that are conditions of high blood sugar linked to low blood pressure and decreased the level of consciousness (Thomas, 2015).

Gestational Diabetes

Gestational diabetes is seen in pregnancies and gestation periods.

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The gestational diabetes impacts on how the cells utilize sugar (glucose). Gestational diabetes leads to high blood sugar affecting a pregnancy and baby's health. The scope of gestational diabetes includes blood sugar returning to normal after delivery. Gestational diabetes infects the ten-year-shorter life expectancies. This is mainly due to the scope of complications that are associated with risks of cardiovascular diseases of including ischemic stroke and heart disease (Pereira, 2013).

Juvenile Diabetes

Juvenile diabetes involves short-acting insulin action through an onset of between 30 minutes and peak action of 4 hours. The intermediate action of insulin onsets in 2 hours. Long-acting insulin is given at bedtime. The action onset is roughly 1 to 2 hours with a sustained action of 24 hours. Juvenile diabetes occurs when beta cells produce insufficient insulin resulting in eventual insulin resistance. Insulin resistance linked to the inabilities of cells to respond adequately to normal insulin levels primarily occurs in the liver, fat tissue, and muscles. The liver shows insulin's normal suppression of glucose release. However, the insulin resistance setting in the liver is inappropriately released through glucose to the blood (Ezrin, & Kowalski, 2011).

Diabetes type II

Diabetes mellitus.....

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